And as I say, most of them really die very horribly, 80 is very bad for you. now that means, that is really bizarre that one should ask the question why should we defy aging.

We also refer to subclinical conditions.

In fact scientist are theorizing that this age proteins may play an important role in the development of atherosclerosis, as well as certain complications of diabetes and chronic kidney failure.

We call it.....make it clear you see that. This is kind of tube over here. Let me draw the blood

Yes. She had multiple sclerosis.

Here's an example from Kevin Eggan's lab at Harvard.

And I developed a chronic disease for which there is no cure.

All of that connective wiring must be insulated with something called myelin.

Neither of them attended university.

In the case of rheumatoid arthritis, the immune system attacks the cartilage around the joints.

This is what we call a plaque.

It would look something like that.

This is one mechanism by which the bacterium Bacteroides fragilis prevents a mouse version of multiple sclerosis.

So over time, I am not going into the details how this happened. It is subject for another video.

And then I wondered was there any relationship between how much people changed their diet and lifestyle whichever group they were in and the changes in PSA?

And the processes that cause this are things like arteriosclerosis for the large vessels or arteriolosclerosis for the small vessels.

As you look at this you will see that the movement energy, I'm going to assume for the two cases is similar, so similar sized bar.

His name is Mike, and he is uniformly impaired on cognition, vision, walking, sensation.

In 2000 when I was diagnosed with multiple sclerosis,

Two things will happen I want to make it clearer relative to the last video, especially since I can zoom in on these arteries two things can happen

I turned to reading the latest research using podmed.gov

Let me just draw this in a reddish color, because you can have blood clots form on the ruptured plaque.

Here are the actual patients, there are 45,000 of them now, sharing their stories as data.

Artery up here, artery over here, and then vein in the other two spots.

His vitals are spiking.

But Beck wasn't dead he heard that, but he was completely unable to move.

And then, when the heart is ready to pump it back out, it goes into this artery, and the name of it is the aorta.

Have the patient take a deep breath and extend neck, and rotate head towards the painful shoulder.

And so we will simulate in that model for your particular cancer and this also will be for ALS, or any kind of system neurodegenerative diseases, things like that we will simulate specifically you, not just a generic person, but what's actually going on inside you.

The motion creates the real time pulsation that is shown visually in a form of light

Now, to start the journey, it's gonna have to out of the left ventricle and into the largest artery of the body.

And let me actually show you where the blood vessels come from. These are called coronary vessels I just write the word coronary here.

It doesn't have any veins.

Coronary vessels So, coronary artery and vein.

And the veins actually drain into a spot , directly into the right atrium.

And on the right you actually see a carotid artery that was engineered.

Mice that have been treated with this bug are more resistant to the disease, as shown by the red line in this graph, and this depends on the activity of a special subset of immune cells, called regulatory T cells, that expresses the marker CD25.

Chronic Fatigue Sundrome.

Remove the ties that bind...

Right it's gonna have to go away from the heart. So that's the aorta. The aorta actually has the little arch like that.

Is it badly infected?

Smelling salts.

If the artery gets nicked, how long do i have?

This, uh,

And actually it stop there because of my arrows, but you can actually see now deoxygenated blood kind of goes from the right ventricle, through the pulmonary trunk, through the arteries and into the left and right lung.